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Paweł P. Liberski

Researcher Next ID · RN-041510

Researcher · Biochemistry, Genetics and Molecular Biology

Medical University of Lodz

Lodz, Poland

Accepting doctoral researchersFunding unknown
Works count
495
Citation count
19,151
H-index
49
i10-index
208

Research interests

Biochemistry, Genetics and Molecular Biology
Medicine
Neuroscience
Nursing
Prion Diseases and Protein Misfolding
Neurological diseases and metabolism
Glioma Diagnosis and Treatment
Alzheimer's disease research and treatments
Trace Elements in Health

Publications

  • Identification of novel risk loci and causal insights for sporadic Creutzfeldt-Jakob disease: a genome-wide association study

    The Lancet Neurology · 2020 · https://doi.org/10.1016/s1474-4422(20)30273-8

  • Cerebrospinal fluid neurofilament light levels in neurodegenerative dementia: Evaluation of diagnostic accuracy in the differential diagnosis of prion diseases

    Alzheimer s & Dementia · 2018 · https://doi.org/10.1016/j.jalz.2017.12.008

  • Germline and somatic FGFR1 abnormalities in dysembryoplastic neuroepithelial tumors

    Acta Neuropathologica · 2016 · 10.1007/s00401-016-1549-x

  • The sheddase ADAM10 is a potent modulator of prion disease

    eLife · 2015 · https://doi.org/10.7554/elife.04260

  • Astrocyte Depletion Impairs Redox Homeostasis and Triggers Neuronal Loss in the Adult CNS

    Cell Reports · 2015 · 10.1016/j.celrep.2015.07.051

  • The toxicity of antiprion antibodies is mediated by the flexible tail of the prion protein

    Nature · 2013 · 10.1038/nature12402

  • Fusion of TTYH1 with the C19MC microRNA cluster drives expression of a brain-specific DNMT3B isoform in the embryonal brain tumor ETMR

    Nature Genetics · 2013 · 10.1038/ng.2849

  • Mutations in SETD2 and genes affecting histone H3K36 methylation target hemispheric high-grade gliomas

    Acta Neuropathologica · 2013 · https://doi.org/10.1007/s00401-013-1095-8

  • Driver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastoma

    Nature · 2012 · https://doi.org/10.1038/nature10833

  • Hotspot Mutations in H3F3A and IDH1 Define Distinct Epigenetic and Biological Subgroups of Glioblastoma

    Cancer Cell · 2012 · https://doi.org/10.1016/j.ccr.2012.08.024

  • Glioma cells showing IDH1 mutation cannot be propagated in standard cell culture conditions

    British Journal of Cancer · 2011 · 10.1038/bjc.2011.27

  • De novo generation of a transmissible spongiform encephalopathy by mouse transgenesis

    Proceedings of the National Academy of Sciences · 2008 · 10.1073/pnas.0810680105

  • Molecular Profiling Identifies Prognostic Subgroups of Pediatric Glioblastoma and Shows Increased YB-1 Expression in Tumors

    Journal of Clinical Oncology · 2007 · 10.1200/jco.2006.07.8626

  • Neuronal cell death in transmissible spongiform encephalopathies (prion diseases) revisited: from apoptosis to autophagy

    The International Journal of Biochemistry & Cell Biology · 2004 · 10.1016/j.biocel.2004.04.016

  • Autophagy is a part of ultrastructural synaptic pathology in Creutzfeldt–Jakob disease: a brain biopsy study

    The International Journal of Biochemistry & Cell Biology · 2004 · https://doi.org/10.1016/j.biocel.2004.04.014

  • Papillary Tumor of the Pineal Region

    The American Journal of Surgical Pathology · 2003 · https://doi.org/10.1097/00000478-200304000-00011

  • Coexistence of Alzheimer-type neuropathology in Creutzfeldt-Jakob disease

    Acta Neuropathologica · 1998 · 10.1007/s004010050870

  • Severe, Early and Selective Loss of a Subpopulation of GABAergic Inhibitory Neurons in Experimental Transmissible Spongiform Encephalopathies

    Brain Pathology · 1998 · 10.1111/j.1750-3639.1998.tb00188.x

  • Heightened expression of tumor necrosis factor alpha, interleukin 1 alpha, and glial fibrillary acidic protein in experimental Creutzfeldt-Jakob disease in mice.

    Proceedings of the National Academy of Sciences · 1996 · 10.1073/pnas.93.18.9754

  • The Original Gerstmann‐Sträussler‐Scheinker Family of Austria: Divergent Clinicopathological Phenotypes but Constant PrP Genotype

    Brain Pathology · 1995 · 10.1111/j.1750-3639.1995.tb00596.x

Current projects

    No projects listed.